nd weakness from the general septic poisoning.

_Treatment._--In treatment the first thing to be sought is the removal
of all offensive and irritant matters from the womb through a caoutchouc
tube introduced into the womb, and into which a funnel is fitted. Warm
water should be passed until it comes away clear. To insure that all the
womb has been washed out, the oiled hand may be introduced to carry the
end of the tube into the two horns successively. When the offensive
contents have been thus removed, the womb should be injected with a
quart of water holding in solution 1 dram permanganate of potash, or, in
the absence of the latter, 2 teaspoonfuls of carbolic acid, twice daily.
Fomentation of the abdomen, or the application of a warm flaxseed
poultice, may greatly relieve. Acetanilid, in doses of half an ounce,
twice or thrice a day, or sulphate of quinia in doses of one-third
ounce, may be employed to reduce the fever. If the great prostration
indicates septic poisoning, large doses (one-half ounce) bisulphite of
soda, or salicylate of soda, or sulphate of quinin may be resorted to.

LEUCORRHEA.

This is a white, glutinous, chronic discharge, the result of a
continued, subacute inflammation of the mucous membrane of the womb.
Like the discharge of acute inflammation, it contains many forms of
bacteria, by some of which it is manifestly inoculable on the penis of
the stallion, producing ulcers and a specific, gonorrheal discharge.

_Treatment_ may consist in the internal use of tonics (sulphate of iron,
3 drams, daily) and the washing out of the womb, as described under the
last heading, followed by an astringent antiseptic injection (carbolic
acid, 2 teaspoonfuls; tannic acid, 1/2 dram; water, 1 quart). This may
be given two or three times a day.

DISEASES OF THE UDDER AND TEATS.

CONGESTION AND INFLAMMATION OF THE UDDER.

This is comparatively rare in the mare, though in some cases the udder
becomes painfully engorged before parturition, and a doughy swelling,
pitting on pressure, extends forward on the lower surface of the
abdomen. When this goes on to active inflammation, one or both of the
glands becomes enlarged, hot, tense, and painful; the milk is dried up
or replaced by a watery or reddish, serous fluid, which at times becomes
fetid; the animal walks lame, loses appetite, and shows general disorder
and fever. The condition may end in recovery, in abscess, induration,
or gangrene, and, in some ca