first moved out from the stable. Our opinion
is asked as to the cause of the lameness, and an inspection is made. With
the changes in the form of the hoof as yet wanting, we have nothing to
guide us, and other causes for the lameness suggest themselves, probably
corns. Evidence of these is not forthcoming, and we in all probability
withhold our opinion until a later visit. On the second or a subsequent
call we are perhaps lucky enough to find our patient down. Diagnosis is
then rendered easier. Made to rise, the animal stands in the attitude we
have described as indicative of laminitis. We have him walked and trotted
out. The symptoms of tenderness disappear, and the animal soon goes fairly
sound. He is, in fact, workable--that is, by anyone who is careless as to
the comfort of his beast.
When following an acute attack, we have the most marked symptoms of pain
and distress, somewhat abating after the second or third week. The walk,
however, is still painful, and, for a short time after rising from the
ground, even difficult.
In short, in both cases we have the horse going on his heels, with a walk
that is painful, and with symptoms of pain that are most apparent when
moved on after a rest.
Later, the changes in the form of the hoof begin to appear. It seems to
have lost its elasticity, and is seen to be dry and chippy, and to have
become denuded of its varnish-like outer covering.
In addition, it is of largely altered shape. The toe, by reason of the
animal walking on his heels, and by reason of an increased growth of horn,
becomes elevated, so that the front of the wall, instead of forming an
obtuse angle with the ground, comes to run very nearly horizontal with
it. The horn of the heels, as compared with that of the toe, takes on an
increased growth. The same thing we have already indicated as happening at
the toe, though in lesser degree. Taken together, this increased growth of
horn at the toe and at the heels has the result of lengthening the diameter
of the foot from before backwards, the transverse diameter remaining more
or less normal. The hoof thus loses its circular build, and comes to
approach nearer an elongated oval.
[FIG. 122.--FOOT BADLY DEFORMED AS A RESULT OF CHRONIC LAMINITIS.]
At this stage, too, the pathological 'ribbing' of the hoof is observable.
The outer surface of the wall becomes marked with a series of ridges
encircling the hoof from heel to heel (see Fig. 81, which illustrates a
|