red and inflamed in appearance.
Later, the abscess--for abscess it is--discharges its contents, the opening
is explored, and we find that in extent it is not confined to the coronary
region, but that it is deep enough to constitute a true sub-horny quittor.
This discharge of the abscess contents may take place at a well-defined
spot on the coronet, or it may ooze out at the junction of the wall
with the skin. In appearance the discharged pus varies. When the softer
structures only are attacked it is thick, and yellow or white in colour;
when bone is involved it is ichorous; and when attacking the horn itself
black or gray. It may or may not be extremely foetid, and often it is
mingled with blood.
When evidence of a previous opening upon the coronet is plain, then it is
not considered wise to attempt a paring of the sole. Instead, poulticing
is at once resorted to, to induce the discharge of the pus through its
original channel. Once this has occurred a fistulous wound remains, which
is open for treatment upon one or other of the lines we shall afterwards
indicate.
COMPLICATIONS--_(a) Necrosis of the Lateral Cartilage_.--This is the
so-called 'cartilaginous quittor' of other writers. In all probability it
is the condition generally understood when the word 'quittor' is used by
one practitioner to the other. Its tendency to keep the disease existing in
a chronic form renders it of grave importance, and for that reason we give
it first mention among the complications.
It may occur as a sequel either of cutaneous or of sub-horny quittor, and
may result either from actual wounding and infection of the cartilage, or
from an attack on it of septic matter originating elsewhere.
Unless there has been discovered a fistula, which on probing is seen to
lead direct to the position in which we know the cartilage to be, we
know of no precise means by which the existence of this condition may be
diagnosed. When free from other complications, the horse with his foot in
this state may travel fairly sound. This is so when the necrosis is situate
in the posterior half of the cartilage, in which case the irritation set up
by the disease is confined to the comparatively non-sensitive tissues of
the cartilage itself and the fibrous mass of the plantar cushion. When
attacking the anterior half of the cartilage, the close contiguity of the
joint renders the disease of a more serious nature. It is then that we have
acute pain, and wit
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