h it extreme lameness, for in this position it is more
than likely that we have involved either the synovial membrane of the
articulation or the tops of the sensitive laminae. It will be remembered
that here the synovial membrane protrudes as a small sac between the
antero- and postero-lateral ligaments of the joint. More or less easily
then it is bound to come into intimate contact with the septic matter
attending the necrosis of the cartilage, and so share in the inflammatory
processes, afterwards communicating them to the interior of the
articulation.

With necrosis of the lateral cartilage is always swelling and thickening of
the skin and subcutaneous structures of the coronet. This is the greater
the longer the disease has been in existence. Upon the swelling is seen the
mouth of the fistula, or it may be the mouths of several, and from them all
a discharge of pus.

The mouth of each fistula is generally filled with a mulberry-like
granulation tissue, standing above the level of the skin, and bleeding
easily if touched. The exuding pus is thin and pale gray in appearance,
gritty to the touch, and generally free from pronounced smell. At other
times its colour is reddened with contained blood, and floating in it are
tiny particles of a pale-green substance, which when picked up and rubbed
between the fingers are seen to be small fragments of the diseased
cartilage.

Should the mouth of a fistula become occluded with the granulations filling
it, and the discharge prevented from escaping, it soon happens that we have
close to the fistula that has closed a tender fluctuating swelling. This
points and breaks, and pus is again discharged from another opening. In
this manner is accounted for the multiplicity of scars and fistulas seen on
the swelling of an old-standing quittor.

The continued, inflammation thus kept in existence has the effect of
rendering the skin and subcutaneous tissues in the neighbourhood greatly
thickened and indurated. This in time leads to a tumour-like enlargement,
and causes the structures of the coronet to greatly overhang the hoof. At
the same time the constant inflammation has made its stimulant effects
noted in a great increase in the growth of the horn of the wall.

Although more abundant, however, the quality of the horn is deteriorated.
The perioplic ring has become obliterated, and the varnish-like appearance
of the healthy wall destroyed. Cracks and fissures in its surface are
nume