ces indicate the near
approach of dissolution,--the face and members become bloated, and the
feet greatly swollen." "The dyspnoea meanwhile, from effusion into the
chest and pericardium, becomes so severe, that the patient cannot
maintain the horizontal position, the expectoration becomes copious,
consisting of a black inky (dintenschwarze), or ash-coloured fluid,
sometimes of mere masses of mucus streaked with black." "The disease is
never accompanied with colliquative sweats or diarrhoea."

I am sorry to find that there is no allusion whatever to the state of
the pulse. Dr Brockmann, in his remarks on the essential nature of this
pulmonary disease of miners, brought under his notice, seems to
entertain the impression that along with the inhaled carbon, resulting
from the combustion of gunpowder, there is also an organic
pigment-deposit present in the pulmonary tissue, which he supposes must
have been formed in the lungs.

I have long entertained the belief, which I have stated in another part
of this essay, that if the carbon is once conveyed into, and established
in the parenchyma of the lungs, that organ commences the formation of
carbon; thus increasing the amount originally deposited. Dr Brockmann
sets forth, as grounds for this view, that "if the parenchyma of the
lungs were filled with carbonaceous dust, their specific gravity ought
to be increased; but this is not the case. A completely melanosed lung
swims in water, both as a whole and when cut into parts." It is very
evident from these remarks, that the author has not seen the disease as
it is exhibited in the third division of morbid action in the collier,
otherwise he would have both observed the lungs considerably augmented
in weight, and also so densely impacted from the accumulation of carbon,
as wholly to sink in water. See for instance case No. 2, where the lungs
weighed about six pounds, and parts of the cellular tissue were so
indurated, as to be cut with difficulty. In this case, the patient did
not expectorate.

Dr Brockmann, as he advances, puts a question here, which more fully
shows that the disease under his consideration was of a mild character
compared with that under our notice. "If," says he, "pulmonary melanosis
arise entirely from inhalation of carbonaceous dust, why is it not
observed in other workmen, who are as much, and even more, exposed to
its influence, as for instance, smelters, or moulders, and colliers?" He
says, further, "we